inflammation, antibody–mediated and cell–mediated immunity.
–Immediate but short–term –nonspecific –started quickly by any event no matter where it occurred.
–provides immediate protection against effects of tissue injury and invading foreign proteins. –inflammatory resp is part of the innate immunity
Infections, trauma, chemical injury, radiation, splinter, virus, fungus
YES!! It is non–specific
– minimize effects of injury or infection – remove damaged tissue – generate new tissue DOES ALL THIS BY DILUTING , DESTROYING, OR NEUTRALIZING harmful agent.
directional movement of WBC to the site of injury, resulting in accumulation of neutrophils and monocytes at the site.
Inflammation is always present with infx but infx is not always present with inflammation
–First leukocyte at the site –55–70% of total WBC –Destroys bacteria by phagocytosis –matured in bone marrow after 12–14d and last only about 12–18 hrs (book 24–48 hr). –Bone marrow releases more to keep up with demand and WBC/neutrophil count goes up. –dead neutrophils , digested bacteria, and cellular debris accumulate and become pus.
–formed as monocytes and mature into macrophages in body tissues. –immediate inflammatory resp and stimulate longer–lasting immune resp. –long life span –early detection between self and non–self cells , protect by phagocytosis –– clear site of debris for healing.
–Early (immediate) reaction of local tissues and blood vessels to injury –Typically occurs before immune response becomes established. –Aimed at removing injurious agent and limiting tissue damage.
vascular response , cellular response , tissue repair